At present, cannabis is only fully legal (on a national level) in Canada and Uruguay. This means that federal research funding for cannabis studies is still difficult for many global research organizations.
Data regarding the relationship between female infertility and marijuana use is limited. However, a study published in the Journal of Women’s Health in 2016 suggests that smoking cannabis can delay a woman’s ovulation by several days.
The general discrepancy in the observations made from these above-referenced studies means we can’t draw any firm conclusions regarding the ongoing debate of marijuana and infertility. Without a doubt, more research needs to be done on the topic.
Additional Research on Weed and Infertility
Another pressing concern – and an ongoing topic of debate circulating for decades – is whether or not marijuana can cause male infertility.
For decades, there has been an ongoing discussion regarding cannabis and infertility. Numerous anecdotal outlets have cited decreased fertility rates among cannabis users (especially a decline in sperm count in males), but is there any clinical or scientific evidence to back up these claims?
Researchers offered several hypotheses attributed to the decline in semen quality. These include:
Furthermore, a separate study published by the National Institute on Drug Abuse suggests that cannabinoids can alter hormone secretion related to reproductive function. Authors suggest that cannabinoids, specifically THC, can “inhibit secretion of LH, FSH, [and] prolactin,” resulting in “decreases in sex steroid hormones [as well as] changes in ovulation.”
Quitting marijuana can be harder than many long-term marijuana users expect, so you and your partner would be wise to quit as soon as possible, while you still have time to get help before getting pregnant. If either or both parents still use marijuana when the baby arrives, you are increasing the risk that your child may use drugs in the future, and parental drug use is implicated in many difficulties for children and families.
Although the link between marijuana and fertility is not straightforward—plenty of marijuana smokers get pregnant and get their partners pregnant—some research has demonstrated that marijuana use can negatively impact you, your partner, or the fertility of both of you.
Obviously, if you are both smoking marijuana, you risk increasing the chances of infertility as a couple.
Furthermore, the effects of marijuana on fertility seem to accumulate over time. This means that although teenage girls who smoke marijuana are more likely to get pregnant, by the time a chronic marijuana smoking woman is in her mid-twenties, she may be more likely to experience a delay in getting pregnant.
Steven Gans, MD is board-certified in psychiatry and is an active supervisor, teacher, and mentor at Massachusetts General Hospital.
Research suggests that marijuana can negatively affect female fertility in the following ways:
Despite the relaxation effects that many people associate with marijuana use, research has shown marijuana has negative effects on the male sexual response.
Clearly, the endocannabinoid system has a role to play in the regulation of processes critical to male reproductive health, such as sperm count, testosterone levels, and levels of other key hormones such as LH.
Use of cannabis may affect human fertility, depending on gender, general health, socioeconomic status and several other factors. Here, we look at existing research on cannabis and human fertility, in order to provide readers with an accurate, up-to-date summary of the current state of scientific knowledge.
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In 2012, the American Society of Andrology published a review of research on the effects of illicit drug use on male fertility. The researchers found that in the majority of studies, it was consistently concluded that cannabis use had a negative impact on male reproductive physiology.
Other studies seen as providing evidence that cannabis use can cause foetal abnormalities are animal studies (Geber & Schramm 1969, Phillipset al, 1971) in which rabbits, hamsters, rats and mice were injected with vast doses of crude cannabis extract (as much as 666mg/kg in one instance!). Such massive doses of cannabis would be practically impossible for a human to consume through conventional means, and are essentially useless as a point of comparison.
And an even earlier study (Kolodny et al, 1974) into testosterone levels in “chronic” cannabis users found that 6 of 17 subjects had oligospermia (low sperm count), and that average testosterone levels in the cannabis-using group were just over half that of the control group. The effect of cannabis on testosterone levels was observed to be dose-dependent.